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If I took a drug that reduced neuronal innervation of my type II muscle fibers, what might I expect to have happen and why

I took a drug that inhibited hepatic (i.e. liver) glucose production.  How might this affect fatigue during long distance aerobic events

Why is it possible for skeletal muscle to grow following resistance exercise even though the glucocorticoid, cortisol, is secreted at higher levels in response to the exercise sessions?

I took a drug that prevented an increase in mitochondrial content in the muscle in response to long term aerobic exercise training.  How would this drug minimize fatigue during exercise and why

If I inhibited the receptor for cortisol (a glucocorticoid released from the adrenal gland) in skeletal muscle, what would likely happen during very prolonged aerobic exercise (2+ hrs)

I took a drug that increased insulin levels in circulation during a bout of aerobic exercise. What is likely going to happen because of this drug

What best describes the change in circulating glucose after completing a very high intensity sprint and why

Which of the following best describes why fatigue can occur even if ATP levels are maintained

Major league baseball players are known to supplement with Testosterone to increase muscle mass and enhance performance.  Why would a baseball player need to supplement with LARGE quantities of testosterone in order to see an effect on muscle mass?

What best describes the theory for why fatigue occurs during a 1 minute sprint at high intensity