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Consider a muscle cell with a mutation in Complex I of the ETC resulting in its complete inactivation. How many ATP molecules will this mutated cell produce per glucose molecule fully oxidized?

The standard reduction potentials ( E’ °) for the half reactions are given ​ Fumarate + 2H++ 2e– → succinate E’° = +0.031 V FAD + 2H+ + 2e– → FADH2 E’° = –0.219 V ​ If succinate, fumarate, FAD, and FADH2 , all at 1 m concentrations, were mixed together in the presence of succinate dehydrogenase, what would happen initially?

Choose the wrong statement concerning the ATPase binding-change mechanism.

Predict the output of the following C program: #includeunion u {    struct{        char a, b, c;    }s;    char string[10];} element;main() {    strcpy(element.string, “Hello!”);// copies one string into another    element.s.b = ‘i’;    element.s.c = ‘?’;    printf(“%.3s, %c”, element.string, element.s.a);}

Suppose you discovered a mutant yeast whose glycolytic pathway was shorter because of the presence of a new enzyme catalyzing the reaction glyceraldehyde 3‑phosphate + H2O + NAD+ ⟶ NADH + H+ + 3‑phosphoglycerate Which of the following would you expect to find in the growth metabolism of the mutant yeast?

Which of the following is not observed in mammalian cells undergoing citric acid cycle?

Which compound transfers reducing equivalents (electrons) into the mitochondrial matrix?

Which chemical prevents the influx of protons through ATP synthase and also is used as an antifungal agent?

Write all the different possible outputs of the following Java multi-threaded program: class Test extends Thread {    private int inc;    public Test(int inc) {        this.inc = inc;    }    public void run() {         for(int i = 0; i < 3;i++)                     Main.x += inc;         System.out.print(Main.x);     } } class Main{     public static int x = 0;     public static void main(String[] args){         new Test(1).run();         new Test(-1).run();     } }

The first control site in the Krebs cycle is inhibited by: