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Which of the following is a characteristic of milia?

A neonate presents after normal vaginal delivery with more than 6 cafe au lait spots measuring greater than 1.5 cm each. The infant should be evaluated for which of the following diseases?

A post-dates infant was born with Apgars 1, 2, 5, and 7 and is currently receiving total body cooling for treatment of hypoxic ischemic encephalopathy. On DOL 3, the infant has had no urine output for 18 hours and the serum sodium is 125. What diagnosis do you suspect?

Match the associated congenital anomalies of the kidneys and urinary tract with their associated presentation and/or management.

A 2 week old preterm infant weighing 900 grams at birth shows pneumatosis intestinalis on an abdominal x-ray. A follow-up film is concerning for potential bowel perforation. Metronidazole (Flagyl) is added to the antibiotic regimen. The goal of the additional antibiotic is to provide coverage for:

Which of the following is true of gonococcal ophthalmia neonatorum?

Teratoma in the neonate under two months of age is malignant in:

A 3520 gram, 38-week gestational age infant is born via spontaneous vaginal delivery to a primigravida. Apgars scores are 7, 9 and 9 at 1, 5 and 10 minutes respectively. The infant breastfeeds well and has had normal meconium stooling. Just prior to discharge at 36 hours of age, the infant has a sudden onset of bilious vomiting. The infant is transferred to the NICU. The physical exam reveals the following: Vital signs: HR 180, RR 70, BP 44/29 (32). Breath sounds are clear and equal. The heart rhythm is regular without a murmur. Pulses are 1+ with a capillary refill time of 4 seconds. The abdomen is distended, tender, and no bowel sounds are heard. He passes a stool with visible bright red blood. An initial abdominal x-ray reveals air in the stomach with an otherwise gasless abdomen. The most likely disorder is:

The most common initial sign of early onset GBS disease with meningitis is:

An infant’s blood type is B positive with a positive DAT, while the mother (who is now G2P1) is blood type O negative. There is no documentation of Rhogam being given to the mother. The indirect bilirubin is elevated and phototherapy is started. What would help you to differentiate between ABO and Rh incompatibility as the cause of the hyperbilirubinemia?