Author: Anonymous

Why is the glutamate hypothesis of schizophrenia difficult to investigate? 

An fMRI study finds overactivity in the amygdala of a person with depression. What might this imply?

In class, we assessed several vignettes describing people who might have a psych disorder. However, there wasn’t a single one that we reached a consensus on (in either direction – yes OR no). Pick two vignettes and answer some questions: Pick a vignette that is the most solid YES for you. Cross-compare to the DSM and decide: is this one really diagnosable, or does the DSM say it’s not? State your final decision and provide a rationale. Pick a vignette that is the most solid NO for you. Cross-compare to the DSM and decide: is this one really not diagnosable, or does the DSM say it could be? State your final decision and provide a rationale. Reflect on both: what are potential benefits and drawbacks (list one for each) of the decisions you made (diagnosing vs not diagnosing) on each of the above vignettes?

How do we know that depression has something to do with serotonin, but isn’t simply low levels of serotonin?

What mechanism of psychedelics like psilocybin and MDMA explain their potential for “re-wiring” traumatic memories in PTSD?

When journaling, which is the most effective strategy for positive growth?

Why is withdrawal from depressants particularly dangerous? 

MAOIs act by:

Which of these is supported by research on sexual orientation? 

I conduct a study in which I measure “state depression.” What am I measuring?